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  • 1 Kilogram/Kilograms .
  • Zhejiang China (Mainland)
  • white
  • shanghai
  • Assay:98%
  • Tag:L-Glutathione 98, white, Oral Liquid
Post Date : September 09
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Item specifics

Zhejiang China (Mainland)
Oral Liquid
L/C,D/A,D/P,T/T,Western Union,MoneyGram
100000 Kilogram/Kilograms per Month .


Molecular Formula C20H32N6O12S2
Molecular Weight 612.63
CAS NO 27025-41-8
EINECS 200-725-4

Molecular Formula C20H32N6O12S2
Molecular Weight 612.63
CAS NO 27025-41-8
EINECS 200-725-4

Standard Enterprise standard

Character Crystalline

Melting point 178-182°C

Assay 98%




lyophilized powder

purified by


total impurities

Ethanol, free

storage temp.


Antioxidant, free radical scavenging, Detoxification, protect the liver, strengthening immune system, Anti-cancer, anti-radiation hazards, anti-allergy.

This groups are reducing agents, existing at a concentration of approximately 5 mM in animal cells. Glutathione reduces disulfide bond formed within cytoplasmic proteins to cysteines by serving as an electron donor. In the process, glutathione is converted to its oxidized form glutathione disulfide (GSSG). 1847859_0_20090704144643.jpg

Company Related ProductsView the Seller's Products
Alpha GPC (powder)

Alpha GPC (powder)

Alpha GPC (powder)

Alpha GPC
Molecular Formula C8H20 N O6 P
Molecular Weight 257.22
CAS NO 283-77-9
EINECS 248-962-2

Chemical name       Alpha GPC

Molecular Formula C8H20 N O6 P

Molecular Weight   257.22

CAS NO        283-77-9

EINECS         248-962-2

Standard      Enterprise standard

Character     White powder or Clearance liquid

Assay           98%


Specification(L-Alpha-GPC Powder, Anhydrous)

Test items



White powder,

Neutral odor, very highly hygroscopic.


Conforms to the standard

Solubility(H2O, 10% w/v)


Bulk Density (Untapped)



Dry and light protected storage;

Stable under normal usage.

Color of solution


Water content




Specific Rotation[α]25D


Related Substances (HPLC)

G.P.E. :<0.2%

Glycerophosric acid: <0.2%

Related Substances(TLC)

Single spot

Purity(by Titration)

L-α-Glycerophosphoryl Choline


Solvents Residue(Ethanols)



Specification(L-Alpha-GPC Liquid)

Test items



Colorless, clear, viscous liquid


TLC: Standard solution and test solution

Same spot, RF

Specific Rotation[α]25D


Heavy Metals(As Pb)






Phosphate Ion

The color of solution should not stronger

Than the control solution(5ppm)



Water Determination


Assay(Anhydrous basis)


L-Alpha Glycerylphosphorylcholine (Alpha-GPC) sometimes called GPC (glycerophosphocholine) is a naturally occurring phospholipid precursor and metabolite derived from soy lecithin.

Medline summary:

Alpha-GPC or GPC has been documented extensively by published studies in both animals and humans to contribute to at least five (5) significant human health functions.

These relevant 'mind-body' activities include, but may not be limited to the following:

1. Increases human Growth Hormone (hGH)

Increases in endogenous human Growth Hormone (hGH) secretion by the anterior pituitary in conjunction with Growth Hormone Releasing Hormone (GHRH); that is, both Alpha-GPC and GHRH act concertedly to stimulate the release of hGH, naturally;


2. Improved mental focus and stimulation of cognitive function

Stimulation of the enzymatic synthesis of phosphatidylcholine (PC) in nerves, muscle cells and all cell membranes, counteracting the age-related decrease in phospholipid (PC) biosynthesis; thus, Alpha-GPC contributes directly to improved mental focus and stimulation of cognitive function;


3. More strength from work-outs and training programs

Acts as a precursor of acetylcholine (ACh); thus, Alpha-GPC activates cholinergic transmission which permits the development of more strength from work-outs and training programs, plus reducing levels of somatostatin in the hypothalamic-pituitary axis;


4. Improved lipotrophic functions in the liver

Elevations in blood and tissue levels of the essential nutrient, choline, which supports improved lipotrophic functions (methyl group transferases) in the liver. Research has shown that fatty liver, a condition associated with obesity, diabetes and heavy alcohol consumption, often leads to cirrhosis of the liver or liver failure. Studies conducted by Alan L. Buchman, M.D., associate professor of medicine at The Feinberg School of Medicine at Northwestern University, have shown that fatty liver can be prevented and possibly even eliminated with increased levels of choline. Alpha-GPC also acts synergistically with the body's store (and/or supplementation) of S-adenosyl-L-methionine (SAM or SAMe) and folic acid, vitamin B12 and vitamin B6 to facilitate methyl group transfers in the brain and liver


5. Improved balanced and coordination

Produces improved balanced and coordination when combined with 'skill set' practice and training as a result of normalized nerve transmission in the brain, and in cardiac, skeletal and smooth muscles.




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3 5 Diiodothyronine

3 5 Diiodothyronine

3 5 Diiodothyronine

3 5 Diiodothyronine
Molecular Formula C15H13I2NO4
Molecular Weight 525.08.
CAS NO. 534-51-0

3 5 Diiodothyronine


Chemical name     2-amino-3-[4-(4-hydroxyphenoxy)-3,5-diiodo-phenyl]-propanoic acid 

Molecular Formula C15H13I2NO4

Molecular Weight  525.08.

CAS NO   534-51-0


3,5-Diiodothyronine is a metabolite of thyroid hormones (thyroxine and triiodo-L-thyronine). It has specific actions and those actions do not involve thyroid hormone receptors. (PMID 15807655)


An intermediate in manuf of thyroxine. Increases serum levels of growth hormone (GH) comprable to T3 Stimulates cytochrome c oxidase - aka decouples Electron Transport Chain (slightly less than 3,3'-T2) Does not interfere with plasma membrane transport of T3 Increases activation of Glucose-6-Phosphate Dehydrogenase


3 5 Diiodothyronine has effects on carriers, ion-exchangers, and enzymes. Recent studies suggest that 3 5 Diiodothyronine may also affect the transcription of some genes


Until recently, 3,5-diiodothyronine (3,5-T(2)) has been considered an inactive by-product of triiodothyronine (T(3)) deiodination. However, studies from several laboratories have shown that 3,5-T(2) has specific, nongenomic effects on mitochondrial oxidative capacity and respiration rate that are distinct from those due to T(3). Nevertheless, little is known about the putative genomic effects of 3,5-T(2). We have previously shown that hyperthyroidism induced by supraphysiological doses of 3,5-T(2) inhibits hepatic iodothyronine deiodinase type 2 (D2) activity and lowers mRNA levels in the killifish in the same manner as T(3) and T(4), suggesting a pretranslational effect of 3,5-T(2) (Garcia-G C, Jeziorski MC, Valverde-R C, Orozco A. Gen Comp Endocrinol 135: 201-209, 2004). The question remains as to whether 3,5-T(2) would have effects under conditions similar to those that are physiological for T(3). To this end, intact killifish were rendered hypothyroid by administering methimazole. Groups of hypothyroid animals simultaneously received 30 nM of either T(3), reverse T(3), or 3,5-T(2). Under these conditions, we expected that, if it were bioactive, 3,5-T(2) would mimic T(3) and thus reverse the compensatory up regulation of D2 and tyroid receptor beta1 and down regulation of growth hormone that characterize hypothyroidism. Our results demonstrate that 3,5-T(2) is indeed bioactive, reversing both hepatic D2 and growth hormone responses during a hypo thyroidal state. Furthermore, we observed that 3,5-T(2) and T(3) recruit two distinct populations of transcription factors to typical palindromic and DR4 thyroid hormone response elements. Taken together, these results add further evidence to support the notion that 3,5-T(2) is a bioactive iodothyronine.




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